All literature
2012Oxidative Medicine and Cellular LongevityReview

The Human Tripeptide GHK-Cu in Prevention of Oxidative Stress and Degenerative Conditions of Aging: Implications for Cognitive Health

Pickart L., Vasquez-Soltero J.M., Margolina A.

Recovery

Editor's note

Pickart's earlier overview emphasizing GHK-Cu's antioxidant signaling and gene-modulation effects relevant to aging tissues. Mechanistic argument that GHK-Cu's age-dependent plasma decline tracks with the same processes it appears to reverse — interesting hypothesis, mostly cell and animal data behind it.

Read the paperDOI  10.1155/2012/324832

Related literature

Adjacent reading.

2021Frontiers in PharmacologyReview

Stable Gastric Pentadecapeptide BPC 157 and Wound Healing

Seiwerth S. et al.

The Sikiric group's consolidated wound-healing review — incisional, excisional, burn, diabetic-ulcer, alkali-burn rodent models lined up in one place. Preclinical only; no human RCT exists. Read it to understand the mechanistic claims, not as clinical evidence.

Recovery
2018Current Pharmaceutical DesignReview

Stable Gastric Pentadecapeptide BPC 157, Robert's Stomach Cytoprotection/Adaptive Cytoprotection/Organoprotection, and Selye's Stress Coping Response: Progress, Achievements, and the Future

Sikiric P. et al.

Sikiric's theoretical synthesis — BPC 157 framed as a mediator of Robert's cytoprotection concept and Selye's stress response. Useful for understanding why the same lab keeps finding the same effect in everything from gut to tendon to brain. Almost entirely self-citation; weigh accordingly.

Recovery
2014Current Pharmaceutical DesignMechanism

Stable gastric pentadecapeptide BPC 157-NO-system relation

Sikiric P. et al.

Sikiric's NO-pathway mechanism paper — BPC 157 modulates nitric-oxide synthesis, competing with both L-arginine and L-NAME. The most-cited mechanistic hook for BPC-157's vascular and healing claims. Single-group work, no independent NO-pathway replication.

Recovery

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